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Across neural cell models, spinal cord injury paradigms, and aged rodent cognition studies, a consistent pattern has emerged: GHK-Cu upregulates neurotrophic factors, suppresses neuroinflammation through NF-kB pathway blockade, and modulates gene expression networks that deteriorate in neurodegenerative contexts

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427 In addition, the mRNA level of HO-1 in the tissues was increased, while the mRNA amount of FtMt was decreased

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