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As a small-molecule inhibitor targeting specific enzymatic activity, it has gained significant attention for its potential influence on adipocyte function, metabolic efficiency, and body-composition dynamics
It was characterized as a selective, membrane-permeable small-molecule inhibitor of nicotinamide N-methyltransferase in biochemical, adipocyte, and mouse research
Radiology 231 , 872879 (2004)
& Working Group on Uric Acid and Cardiovascular Risk of the Italian Society of Hypertension (SIIA)
These findings underscore lipid peroxide accumulation as a hallmark of ferroptosis, and suggest that downregulating ACSL4, LPCAT3, LOXs, or POR can suppress PUFA-PL-OOH formation and effectively inhibit ferroptosis
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