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GPX4 requires GSH as a cofactor to inhibit lipid peroxidation, and GPX4 has gained research momentum because of its important role in maintaining spermatogenesis [77]
a Pilot Study
This phase typically provides the clearest indication of whether the protocol is working for a given individual and condition
Treatment with FOXO4-DRI at 25 M in serum-limited senescent fibroblast and keloid organ culture models promoted apoptosis, decreased G0/G1 phase cells, and induced p53-pS15 nuclear exclusion confirmed by Annexin V-FITC/PI apoptosis assays and TUNEL staining of keloid organ cultures at days 3, 7, and 10
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