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Through stimulation of pro-opiomelanocortin (POMC) neurons while simultaneously inhibiting neuropeptide Y/agouti-related peptide neurons in the arcuate nucleus, GLP-1 receptor agonism promotes satiety, reduces meal size, and decreases overall caloric intake [35]

Clinicians have separate roles

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References Gwyer, D., Wragg, N

Well, so that goes back to what I, we were saying about deficiency is if there is a physiologic deficiency, then we need a physiologic dose