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The neuroprotective properties of GLP1R agonists correlate with their ability to cross the bloodbrain barrier

The decline in the activity of mitochondrial and peroxisomal isoforms points toward the fungus-promoted precocious senescence that led to the disease development (Kuzniak and Skodowska, Sesamum orientale plants, upon infection with the fungus Alternaria sesami displayed an initial increase in the activity of APX, MDHAR, and GR followed by a gradual decrease in the corresponding activities (Shereefa and Kumaraswamy, 2014)