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These findings suggest that GLP treatment is associated with higher resting cardiovascular activation and reduced parasympathetic activity , indicating increased physiological load reported across studies[1]

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The authors argue that . For example, one of the authors, Paul Ridker of Harvard University Brigham and Women's Hospital has led research that found that: 1 downstream biomarkers of inflammation such as high sensitivity C-reactive protein (CRP) and interleukin-6 (IL-6beta) are associated with an increased risk of CV events, independent of the cholesterol level (Ridker P

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Jiang, X., Stockwell, B