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Since the liver plays a central role in the interorgan GSH homeostasis, liver dysfunction might affect endogenous production and utilization of GSH, and possibly further lead to dysregulation of the GSH-dependent antioxidant system
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Both work by targeting specific receptors involved in appetite regulation, glucose metabolism, and energy balance, but they do so through distinct hormonal pathways
N -homocysteinylation induces different structural and functional consequences on acidic and basic proteins
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