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Using targeted proteomics, ENPP-2 was increased in cerebrospinal fluid (CSF) of AD patients with amnestic mild cognitive impairment (aMCI), and ENPP-2 directly reflects the brain glucose steady state (42, 43)
Because amylin and GLP-1 stimulate different receptors, their effects complement and add up more strongly than either pathway alone
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But others (like calcium and iron) jockey for position on the same metabolic pathways, effectively cancelling each other out
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