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The autophagy modulator trehalose has been shown to facilitate the clearance of dysfunctional mitochondria and suppress aberrant trypsinogen activation
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Users who stop early or use the tablets inconsistently are the most common source of "it doesn't work" feedback, when the actual issue is timeline mismatch with biology
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Accumulating data point to an essential role for ROS as mediators in the activation of ATG under several stimuli (starvation, pathogens, death receptors), even though the nature of this involvement remains still elusive
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