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Harwaldt P, Rahlfs S and Becker K
The triple-overlap signature prominently enriched for lipid metabolism genes ( LPL, FABP5, PLIN2, ABCA1, COLEC12 ), reflecting the central role of DAMs in supporting lipid handling following demyelination
These treatments decreased cell viability and increased both soluble and lipid reactive oxygen species (ROS) generation but did not affect mitochondrial ROS or mitochondrial mass
Absence of Gclc impairs mitochondrial respiration in FoB Our data above showed that glucose-derived fluxes into the TCA cycle were reduced in GSH-deficient FoB
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