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Specifically engineered for higher biological potency in vitro, IGF-1 LR3 has an increased half-life and a binding aversion to native proteins within the body that make it ideal for both research and large-scale culturing
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Heparin binds to a specific site on antithrombin III, producing an altered conformation of the protein, and the new conformation has a higher affinity for thrombin as well as its other substrates
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