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Interestingly, in G6PD-deficient infants these conditions are poorly correlated with hemolysis and the presence of Heinz bodies

This increase in norepinephrine stimulates -adrenergic receptors in the brains satiety center, inducing a stress-response signal that diminishes hunger

Background Inflammatory bowel disease (IBD) is an idiopathic condition characterized by chronic relapsing inflammation in the gastrointestinal tract

Therefore there is an unmet need for therapeutics which can result in a clinically meaningful reduction in ICP without side effects which compromise cognition

For example, research suggests GLP-1s might impact reward circuits in the brain to influence behavior