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Transfer of liraglutide from blood to cerebrospinal fluid is minimal in patients with type 2 diabetes

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-Glutamylcysteine is a GPx1 cofactor that detoxifies H 2 O 2 To decipher how -glutamylcysteine detoxified ROS but otherwise avoiding the influence of cytosolic GCL, we expressed a silent mutant mito GCL form ( mito GCL (mut)) refractory to the action of a small hairpin RNA against GCL (shGCL) 5 , in HEK293T cells (Fig

Ros-triggered endothelial cell death mechanisms: focus on pyroptosis, parthanatos, and ferroptosis

Interestingly, the neural circuits responsible for the metabolic effects of GLP-1R agonism may be distinct from those mediating nausea or malaise (Huang et al., 2024), suggesting that future therapies could be tailored to preserve metabolic benefits while minimizing gastrointestinal side effects