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The proposed pathway is restoration of pinealocyte function either by direct gene-regulatory effects on serotonin-N-acetyltransferase (AANAT) and related enzymes, or indirectly by reducing neuroendocrine stress signalling

For example, ApoE4 has been shown to promote the nuclear translocation of HDACs in human neurons, resulting in decreased BDNF expression [255]

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C3 transferase-expressing scAAV2 transduces ocular anterior segment tissues and lowers intraocular pressure in mouse and monkey

the control only reached 35 % When they blocked VEGFR2 (with the selective inhibitor SU5416), all of these effects disappeared Why it matters: The last point is what holds the study together