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Selective GH release without significant cortisol or prolactin increases [3]

This could enable the treatment of hypoxic tumors, which represent a major challenge in anticancer drug development

Preclinical studies have found that Ex-4 significantly upregulates Sirt3 expression in glioblastoma by activating the GLP-1R, which subsequently inhibits the growth, migration, invasion, and epithelial-mesenchymal transition of glioma cells [87]

Summar ML, Mew NA (2018) Inborn errors of metabolism with hyperammonemia: urea cycle defects and related disorders

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