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The LR3 (Long R3) modification is a clever bit of bioengineering that dramatically extends its half-life and, crucially, prevents it from being bound and rendered inactive by insulin-like growth factor-binding proteins (IGFBPs) in the bloodstream
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Madsbad, S
Furthermore, increasing attention has been directed toward using MSC-Exos to inhibit ferroptosis in cardiovascular diseases (Table 1)
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