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induces oxidative damage in the rats [25]

And all it did was survive

GSH inhibits PSC activation induced by high glucose via the ROS/TGF/SMAD pathway in vitro The above results indicated an increase in pancreatic fibrosis in rats treated with LOsG, which was accompanied by an increase in ROS/TGF/SMAD signalling, and PSC proliferation and activation, which were all prevented by GSH

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Differential Diagnosis The differential diagnosis primarily includes CALM, congenital melanocytic nevus, plexiform neurofibroma, and congenital smooth muscle hamartoma