GLP-1, a hormone primarily secreted from the ileum in response to nutrient intake, exerts multiple metabolic effects including appetite suppression, delayed gastric emptying, enhanced insulin secretion, and inhibition of glucagon release [4]
[13] [14] GLP-1 receptor agonists are not classified as stimulants and do not directly increase energy expenditure in a clinically meaningful way
GSH is synthesized from cysteine, glutamate, and glycine by the ATP-dependent enzyme -glutamyl-cysteine (-GC) ligase (GCL) synthetase, which is composed of the -GC ligase catalytic subunit (GCLC), the -GC modifier subunit (GCLM), and GSH synthetase (GSS) 24
But we didnt stop there
Neutrophil and Phagocytic Dysfunction Neutrophil chemotaxis, phagocytic capacity, and oxidative burst precision are all significantly impaired in Type 2 Diabetes