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These findings underscore the potential of glycitin as an anticonvulsant agent, elucidating its mechanism of action through histological protection, modulation of oxidative stress markers, and activation of the Nrf2/HO-1 signaling pathway

Viral infection reprograms host genomics The SARS-CoV-2 protein, nsp1 , binds to human ribosomes and inhibits host cellular translation 33

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Incidence but not intensity increased with the duration of use, and the change was irreversible