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Notably, many of these non-motor symptoms appear years or even decades before the beginning of overt motor indications, indicating the possibility of serving as early prodromal biomarkers and mirroring the systemic, multisystemic aspect of PD pathogenesis [4]

Several signaling pathways in response to ANG II are mediated by reactive oxygen species (ROS) ( in vitro GLP-1 inhibited the ANG II-induced mesangial cell damage by suppressing superoxide-mediated nuclear factor-kB activation

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