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Both compounds are researched for their potential roles in conditions associated with mitochondrial dysfunction, oxidative stress, and metabolic disturbance, including cardiovascular disease, diabetes, and neurodegenerative disorders

Deletion of the fibronectin gene from mouse LECs did not affect the initiation of TGF signaling at 48 hours PLI, but it blocked sustained TGF signaling at 72 hours PLI and beyond, greatly reduced injury-induced LEC proliferation, and the assembly of fibrotic ECM ( Figure 4 ) (185)

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