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Via TGR5, UDCA inhibits ERK1/2 phosphorylation, blocking the non-canonical TGF /WWP2 pathway to downregulate fibrosis-related genes (COMP, FOXP2) and reduce collagen I/VI synthesis (Reilly-O'Donnell et al., 2024)

As IBD involves oxidative stress and inflammation, these diverse antioxidants collectively act as a shield against cellular damage, offering a multifaceted approach to treatment

We fed a culture of gut microbes with l-carnitine and measured the TMA that the microbes produced

Furthermore, CRIP2 depletion and copper-induced CRIP2 degradation increase reactive oxygen species (ROS) levels and activate autophagy in H1299 cells

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