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After coronary artery ligation in mice, intraperitoneal T4 treatment upregulated cardiac ILK and Akt activity, enhanced early myocyte survival, and improved cardiac function establishing the mechanistic basis for the cardiac and broader tissue-repair claims associated with thymosin beta-4 and its 1723 fragment TB-500

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Treatment with IGF-1 could accelerate cell cycle progression from G 0 /G 1 to S phase, whereas this progression was inhibited by the presence of LY294002 or BAY11-7082

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