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J Biol Chem 2006
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Evidence for a role for oxidative stress in schizophrenia includes studies reporting reduced glutathione blood levels [9], reduced post-mortem glutathione concentrations [10, 11], and genetic evidence implicating schizophrenia-related polymorphisms in genes involved in glutathione synthesis [12]
We found that component anwuligan in forsythia may activate the Wnt/-catenin signaling pathway by binding to CTNNB1
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