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A new mechanism involving ERK contributes to rosiglitazone inhibition of tumor necrosis factor-alpha and interferon-gamma inflammatory effects in human endothelial cells
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It promotes energy and fat metabolism, and is found naturally in cells which use fat for energy
Defective autophagy heightens TECs susceptibility to ferroptosis in DKD, demonstrating the close correlation between autophagy flux impairment and ferroptosis (79)
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