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The accumulation of amyloid plaques is a pivotal factor associated with subsequent neuronal toxicity in AD pathogenesis, eventually associated with synaptic dysfunction and more severe cognitive deficits [38]
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It catalyzes the conversion of H 2 O 2 to water and molecular oxygen in two steps [59, 83, 84]: Catalase-Fe (III) + H 2 O 2 compound I Compound I + H 2 O 2 catalase-Fe (III) + 2H 2 O + O 2 The presence of bound NADPH in each subunit may help protect the enzyme from being inactivated by H 2 O 2 [83]
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