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Interpretation: absence from labelling does not prove impossibility
They: Produce collagen Organize extracellular matrix Restore tensile structure In experimental tendon transection models, BPC-157 has been associated with: Increased fibroblast density Enhanced migration into injury site Accelerated granulation tissue formation Proposed signalling involvement includes: FAK (Focal Adhesion Kinase) activation ERK1/2 signalling Src pathway modulation FAK signalling is particularly important because it regulates: Cell adhesion Cytoskeletal reorganization Migration along collagen scaffolds Tendon strength depends not just on collagen quantity but alignment
Usually kills the sweet tooth, that works quite well
Figure 1 below depicts the incretin effect in a normal patient versus a diabetic patient
Patients taking insulin or sulfonylureas alongside GLP-1 therapy should monitor for hypoglycaemia when reducing sugar intake and seek medication review as needed
