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doi: 10.1038/nrn1824 52

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The aforementioned pathological changes during the ischemic and reperfusion phasesincluding calcium overload, mitochondrial dysfunction, oxidative stress surge, inflammatory cascade activation, cell death network formation, and microcirculatory dysfunctionare systematically visualized in the schematic diagram, which clearly depicts e sequential progression and interconnections of these mechanisms (Figs

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Second, the inability to quantify central nervous system (CNS) GSH concentrations in vivo has substantially hindered therapeutic trials targeting CNS augmentation

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Glut1 exacerbates trophoblast ferroptosis by modulating ampk/acc mediated lipid metabolism and promotes gestational diabetes mellitus associated fetal growth restriction

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Understanding body temperature changes and immune responses

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