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POTENTIAL MECHANISMS UNDERLYING INFECTION-ASSOCIATED DYSAUTONOMIA & POTS The autonomic nerves or autonomic ganglia may be affected by: Chronic infection Autoimmune response triggered by an infection Damage to autonomic nerves by an infection Neuro-inflammation triggered by debris left-over after an acute infection is treated CHRONIC ILLNESSES ASSOCIATED WITH POTS Post Treatment Lyme Disease Long COVID Myalgic Encephalomyelitis/Chronic Fatigue Syndrome or ME/CFS Sjogrens Syndrome Systemic Lupus Erythematosus Hashimotos Thyroiditis Celiac Disease Diabetes MS Other Infection Associated Illnesses RESEARCH NEWS Review Article by Brit Adler, MD, in Frontiers in Neurology: Dysautonomia following Lyme disease: a key component of post-treatment Lyme disease syndrome

Liposomal formulations encapsulate glutathione in phospholipid vesicles, potentially allowing greater intestinal absorption and cellular uptake

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Clinically, treatments targeting different aspects of nicotine addiction, such as reinforcement, withdrawal, and cue-associated learning ranging from complete cessation to substitution with less harmful products are enforced accordingly

Therefore, even GSH generated from GSSG is not masked