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If you answered yes to all three, continuing treatment is reasonable

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Exendin-4 (an endogenous insulin releasing incretin, GLP-1) reduces A accumulation and tau hyperphosphorylation in cellular and animal models of AD (85, 151, 153156)

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And because AMPK, IGF-1, mTOR, and the sirtuin pathways all interlock with GLP-1, the relevance of SG to reproduction rightly draws notice

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