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Jpn Circ J 47:13911397
The NAD(+)-dependent SIRT1 deacetylase translates a metabolic switch into regulatory epigenetics in skeletal muscle stem cells
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Supports metabolic flexibility in both fasted and fed states Useful for individuals with insulin resistance or post-cycle metabolic slowdown May complement fasting, low-carb, or Clean Carbs strategies for performance athletes MOTS-c administration significantly improved glucose tolerance and insulin sensitivity in aged and high-fat-fed mice. Lu et al., Aging Cell Promotes Fat Loss Without Muscle Breakdown By activating AMPK and mitochondrial uncoupling, MOTSc encourages the body to burn fat more efficiently especially during caloric restriction or training
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