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The protein-membrane binding simulations revealed that, for both AtSNX1 and MmSNX1, the interaction with the membrane involved a gradual decrease in the distance between the protein molecules and the membrane, along with the insertion of the AH domain into the membrane (Fig
[25] The form of plasticity known as long-term potentiation takes place at glutamatergic synapses in the hippocampus, neocortex, and other parts of the brain
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