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Mechanistic studies have demonstrated that 5(S),15(S)-DiHETE-mediated ferroptosis signalling facilitates the degradation of hypoxia-inducible factor-1 (HIF1)

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References: Blair E, Miller A

TSS pathway activity is suppressed by excess hemin under hypoxia, and the suppression is reversible through DMF treatment The afore findings suggest that supplementing cystine at physiological concentrations is a prerequisite for investigating the function of CBS in SCD erythroblasts