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These nanoparticles can break the thioester bond in an IRI-induced ROS-rich environment, releasing the anti-inflammatory peptide HDM

Following the KEAP1 inactivation, the expression and the nuclear translocation of nuclear factor-erythroid 2 p45-related factor 2 (NRF2) are increased, which subsequently promotes the transcription of antioxidant proteins like superoxide dismutase 2 (SOD2) and heme oxygenase 1 (HO-1), and leads to the mitigation of synovial inflammation

This is where genetics comes into play: Converting the forms of B6 we get in our foods into the active P5P form utilizes specific genes

P.ChiewA.Abdel-RahmanS