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Through stimulation of pro-opiomelanocortin (POMC) neurons while simultaneously inhibiting neuropeptide Y/agouti-related peptide neurons in the arcuate nucleus, GLP-1 receptor agonism promotes satiety, reduces meal size, and decreases overall caloric intake [35]
Ro leans more on an asynchronous model
Michael Galitzer: Absolutely
2
They regulate key biological processes such as inflammation, cellular migration, proliferation, and angiogenesis [19, 20]
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