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Our preclinical findings demonstrate promising efficacy, with FASN inhibition significantly ameliorating diabetes-induced liver injury in a rodent experimental model. This provides further evidence for the weight-independent metabolic effects of semaglutide in managing MASH
[1] [2] The medication works by mimicking the action of the naturally occurring hormone GLP-1, which stimulates insulin secretion in response to elevated blood glucose levels, suppresses glucagon release, and slows gastric emptying
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The values are expressed as the number of cells/min/100 m
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