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Biochim Biophys Acta 1831(2):291299
Hohenstein B, Daniel C, Wagner A, Stasch JP, Hugo C (2005) Stimulation of soluble guanylyl cyclase inhibits mesangial cell proliferation and matrix accumulation in experimental glomerulonephritis
Phase 1 data Phase 1 studies established the basic pharmacokinetic profile, confirming the extended half-life predicted by the structural design and target engagement at the three receptors
EZH2-mediated H3K27me3 methylation (requiring SAM) at the promoters of senescence-promoting genes keeps those genes silenced while SIRT1 and FOXO3 activate longevity-promoting gene sets
As well, KYNA-induced inhibition of 7nAChRs reduces presynaptic glutamate release, thereby indirectly dampening AMPAR activation, a mechanism that may protect neurons from glutamate-driven excitotoxicity under pathological conditions [82]
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