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L., Eckerson, J
Our results herein showed that rotenone treatment triggered a robust decrease of LC3-II, probably through inhibition of autophagy, which aggravated deposition of aberrant proteins and subsequent cell death 53
However, in diabetic wounds, the transition from M1 to M2 is disrupted, leading to the accumulation of M1 macrophages, which produce ROS and sustain long-term inflammation
Immunofluorescent staining was performed to analyze the expression of M2 gene markers
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