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PMC+1 Anti-inflammatory and cytoprotective actions across multiple injury models

Treadmill exercise rescues mitochondrial function and motor behavior in the CAG140 knock-in mouse model of Huntingtons disease

These differences specifically included metabolic genes, including those involved in the incomplete denitrification pathway, information processing genes, as well as genes known to be involved in host damage, and numerous LOS biosynthesis genes

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15 however, when there is a disproportionately high amount of ROS, it induces oxidative tissue damage