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Genomic and epigenomic aberrations in esophageal squamous cell carcinoma and implications for patients
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Taken together, these findings suggest that APR-246 could induce tumor cell death through both reactivation of mutant p53 and inhibition of cellular thiol-dependent redox system proteins (GPX4, TXNDR1, GRX1), providing novel combinatorial strategies for AML therapy
GPX3, similar to GPX1, is located at a low level of the selenoprotein level and is sensitive to selenium content in the body, so it is often used as a biological marker to measure selenium content in the body
Cognitive impairments are also widespread, ranging from subtle executive function deficits to severe disorientation, with minimal hepatic encephalopathy present in up to 80% of cirrhosis patients
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