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Specifically, leukocytoclastic vasculitis and TNF alpha inhibitor-induced lupuslike syndrome (TAILS) will be discussed below

[8] Those data are important for target biology in neuropathic-pain systems, but they do not establish LanCL1 as the mediator of adipocyte lipolysis, fat oxidation, or body-weight effects

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A primary pathway is the Nrf2/ARE antioxidant defense system