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This mechanism is the foundation of its documented ability to stimulate keratinocyte, endothelial cell, and stem/progenitor cell migration to sites of tissue damage in pre-clinical models

Additionally, by reducing the accumulation of toxic byproducts, it protects against cellular damage and supports overall health

Sickle cell disease

The pharmacokinetics suit a once-weekly rhythm, with an approximate six-day half-life confirmed in the early phase 1b work

654,655 Perls staining and measurements of non-hemoglobin iron revealed iron overload in the ipsilateral cortex of TBI mice and brain microvascular endothelial cells (BMECs)