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GLP-1 may induce sodium excretion by blocking sodium reabsorption in the proximal tubule through inhibition of sodium-hydrogen exchanger isoform 3 (NHE3) activity [23], as no GLP-1 receptors appear to be present in human kidneys outside of the renal vasculature [19]
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In some ways, the failure of the first generation of PPAR agonists paved the way for the Ozempic revolution
Apoptosis and clearance of apoptotic cells
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