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This result could be repeated in two other datasets of GC, GSE15459 and GSE27342 (Fig

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Intra-VTA exendin-(9-39) pretreatment blocked the ability of CNO to attenuate cocaine seeking (Fig 3E & 3F) indicating that the suppressive effects of activating endogenous NTSVTA circuits on drug seeking depend, in part, on enhanced GLP-1 signaling in the midbrain

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Its molecular modification enhances resistance to enzymatic degradation, resulting in a longer half-life compared to earlier GLP-1 analogues