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Neuroprotective Effects of Glucose-Dependent Insulinotropic Polypeptide in Alzheimers Disease

Our study was motivated by the previous observation that mitochondrial muscle diseases cause a secondary NAD + deficiency in the skeletal muscle 15

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40 Preclinical studies have shown that these effects are mainly mediated trough the effects of GLP-1R activation in the central nervous system (CNS) by the modulation of orexigenic and anorexigenic signaling pathways