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Under normal conditions, GPX4 utilizes GSH as a co-substrate to reduce (P)LOOH to non-toxic lipoalcohols ((P)LOH), inhibiting the occurrence of ferroptosis [38]
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Although many contradictory results and conclusions can be attributed to lack of understanding of the established mechanisms, methodological problems and misinterpretation of data, there are many legitimate ways the signaling mechanism of injury can be affected resulting from minor modifications up to major changes including a complete switch of the mode of cell death
Costa I, Barbosa DJ, Benfeito S, Silva V, Chavarria D, Borges F, Remio F, Silva R (2023) Molecular mechanisms of ferroptosis and their involvement in brain diseases
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