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Arch Toxicol 94(11):3799
Long-term safety concerns are lower than for compounds that introduce entirely foreign mechanisms
Chronic hyperglycemia initiates a cascade of molecular eventsincluding the accumulation of advanced glycation end products (AGEs), activation of the polyol pathway, enhanced protein kinase C (PKC) signaling, and mitochondrial dysfunctionculminating in glomerular hyperfiltration, podocyte injury, and progressive glomerular and tubulointerstitial fibrosis
The key question: What happened to patients during the first six months of after diagnosis
No, it is not approved for therapeutic or clinical use
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