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We suggest that loss of cuproprotein function is at the core of ALS pathology, a condition that is driven by a combination of unbound copper and ROS that can either initiate and/or accelerate protein aggregation

reported that BPC157 promotes angiogenesis via VEGFR2 upregulation [16], how BPC157 interacts with VEGFR2, whether it physically binds, and the mechanism underlying VEGFR2 upregulation remain unclear

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Overexpression of -Syn, cytosolic acidification, and Ser129 phosphorylated pathogenic -Syn could all induce translocation of -Syn to mitochondria (Cole et al., 2008

Your IV drip is running, and suddenly your arm feels cold, your stomach turns, or the needle site starts to ache