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Our results demonstrate that METTL3 is upregulated in the aortas of TAAD patients, and METTL3 overexpression facilitates ferroptosis of HASMCs by promoting the mRNA degradation of SLC7A11 and FSP1, then reducing their protein levels

In a healthy thyroid, selenium-dependent enzymes (specifically glutathione peroxidases) act as a cleanup crew, neutralising the hydrogen peroxide before it can damage the delicate thyroid tissue

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