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In RCCs that were either ETC or TCA cycle deficient, it was found that cancer cells relied on the reductive carboxylation of glutamine-derived citrate to produce acetyl-CoA and other precursors to TCA cycle metabolites

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Blocking the Notch pathway or the NF-B pathway tended to counteract the pro-angiogenic implications of TB-500 in both cell lines and murine models

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Following the study period, the femurs of the murine models were analyzed using mid-diaphyseal peripheral quantitative computed tomography (pQCT) scans